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. 1999 Jun;73(6):4543–4551. doi: 10.1128/jvi.73.6.4543-4551.1999

FIG. 6.

FIG. 6

Summary of the behavior of the panel of Z(S186) mutants. (A) Hypothetical model for behavior of ZEBRA mutants on Rp. ZEBRA mutants that are unable to activate Rp have two possible defects: a low affinity of binding to Rp and an inability to be phosphorylated. (B) Hypothetical model for capacity of ZEBRA mutants to synergize with Rta on the BMRF1 promoter. Synergy requires mutual contact between Rta, ZEBRA, and protein X. ZEBRA mutants that do not synergize with Rta are unable to contact protein X.