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. 2024 Jul 16;12:118. doi: 10.1186/s40478-024-01798-9

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — A, B Patient #2 (15 y/F) presented with an intraventricular diffuse low-grade glioma located at the lateral ventricle, exhibiting heterogeneous high signal intensity on contrast-enhanced T1-weighted and T1-fluid-attenuated inversion recovery (FLAIR) images. B The tissue in the H&E section resembled a myxoid glioneuronal tumour, characterized by monotonous round cells within a myxoid background. D, F Immunohistochemical analysis revealed positivity for GFAP, TRK, and Olig2 in this tumour. G An Arriba plot generated from next-generation sequencing (NGS) data using RNA identified the KIF5A::NTRK2 in-frame fusion in the tumour (C, D: H&E, E: GFAP, F: Olig2; scale bar, C-E: 200 μm, F: 100 μm)