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. 2024 Jul 16;16:91. doi: 10.1186/s13148-024-01709-8

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Decreased H3K27ac accumulation attenuated Dox-induced apoptosis in cardiomyocytes. A, B Representative western blot (A) and statistical data (B) showing the levels of BCL2 from Dox-treated cardiomyocytes with or without C646 for 24 h. n = 6 per group. C, D Representative western blot (C) and statistical data (D) showing the levels of cleaved caspase3 from Dox-treated cardiomyocytes with or without C646 for 24 h. n values are 8, 9, 9, 8, respectively. E, F Representative images (E) and statistical data (F) of TUNEL-positive cells (red) from Dox-treated cardiomyocytes with or without C646 for 24 h. n = 6 per group