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. 2024 Jul 3;15:1404119. doi: 10.3389/fphar.2024.1404119

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Copyright © 2024 Liu, Zhou, Wu, Chen, Zheng, Wang, So, Ma, Wang and Chiu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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LRE mitigated oxidative stress in the retina of the laser-induced COH and the DBA/2J mice. (A) Representative images of 8-OHdG stained retinal sections from normal, water-fed, and LRE-fed COH mice. Scale bar, 25 µm. (B) LRE significantly reduced the fluorescence intensity of 8-OHdG in the retina of the COH eyes. (C) The gene expressions of HO-1, SOD-2, and GPX-4 in the retina of the normal, water-fed, and LRE-fed COH eyes. (D) Representative images of 8-OHdG stained retinal sections from the C57BL/6J control, water-fed, and LRE-fed DBA/2J mice. Scale bar, 25 µm. (E) LRE significantly reduced the fluorescence intensity of 8-OHdG in the retina of the DBA/2J mice. *, p < 0.05; **, p < 0.01; ***, p < 0.001.