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. 2024 Jul 17;22:666. doi: 10.1186/s12967-024-05344-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Experimental Design. (A) Scheme depicting the workflow used in this study. Sera were collected from SCI patients with no (NR) or strong (SR) recovery and depleted from high and medium abundant proteins for subsequent tagging and detection of low abundant proteins using mass spectrometry. After enrichment analysis (NR vs. SR), some of these proteins were further validated by ELISA. (B) Box plot showing the distribution of SR (green) and NR (red) patients studied according to their Integrated Neurological Change Score (INCS) between 30 and 120 days after injury. (C) Distribution of SR (green) and NR (red) patients according to their AIS grade conversion (-1 to + 3) between 30 and 120 days after injury