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. 2024 Jul 20;43:200. doi: 10.1186/s13046-024-03116-6

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 9 — Circ_0007386/miR-383-5p/CIRBP axis correlates with NSCLC prognosis and the schematic diagram illustrates the mechanism. A-C. Kaplan–Meier survival plots for NSCLC patients based on low and high expressions of circ_0007386, miR-383-5p, or CIRBP (n = 80). D-F. Correlation analysis of circ_0007386, miR-383-5p, and CIRBP expression analyzed using RT-qPCR in NSCLC tissues (n = 80). G. Schematic representation of the underlying mechanism where hypoxia-enhanced YAP1-EIF4A3 interaction mediates circ_0007386 circularization and promotes non-small cell lung cancer progression via miR-383-5p/CIRBP axis. Data are shown as means ± SD. *p < 0.05, **p < 0.01