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. 2024 Jul 10;25(14):7554. doi: 10.3390/ijms25147554

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© 2024 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Crosstalk between senescent and immune cells in fibrosing interstitial lung diseases. The biological disturbances in senescent cells enable them to engage in complex communication with the host immune system. Various immune cell populations actively participate in the surveillance and regulation of these senescent cells, highlighting the intricate interplay between cellular senescence and immune responses in pulmonary fibrosis. NK: Natural Killer; NKG2: natural killer group 2; MICA: MHC class I polypeptide-related sequence; HLA-E: Major Histocompatibility Complex; Class I, E, CALR: Calreticulin; CD47: Cluster of Differentiation 47; CD24: Cluster of Differentiation 24. Figure created using BioRender (https://biorender.com), accessed on 7 July 2024.