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. 2024 Jul 15;16:1421841. doi: 10.3389/fnagi.2024.1421841

Table 2.

Overview of variants in the KIF1A gene identified in ALS patients.

ID Position (hg19) Refseq ID cDNA change Protein change dbSNP Minor allele frequencies Functional predictions ACMG
gnomAD_genome_ALL gnomAD_exome_ALL SIFT Polyphen2 MutationTaster MetaSVM MetaLR ClinPred M_CAP CADD Pathogenic (total) Evidence Classification
P1 chr2:241710533 NM_001330290 c.1271C>T p.Pro424Leu rs1254343314 PM2 VUS
P2 chr2:241696840 NM_001244008 c.2753_2754insGGA p.Asp918delinsGluAsp rs758125020 1.68E-02 1.74E-02 PM2, PM4 VUS
P3 chr2:241689888 NM_001244008 c.2935G>A p.Glu979Lys rs764324827 4.00E-06 T P D T D D D 35 6 (8) PM2, BP4 VUS
P4 chr2:241685282 NM_001244008 c.3247G>A p.Ala1083Thr rs201793635 4.00E-04 3E-06 T B P T T T D 5.97 1 (8) BP4 VUS
P5 chr2:241685282 NM_001244008 c.3247G>A p.Ala1083Thr rs201793635 4.00E-04 3.00E-04 T B P T T T D 5.97 1 (8) BP4 VUS
P6 chr2:241683410 NM_001244008 c.3533 T>C p.Phe1178Ser - D D D 25.6 3 (3) PM2, PM1, PP3 VUS
P7 chr2:241680755 NM_001244008 c.3680C>T p.Pro1227Leu rs374244985 2.00E-04 2.00E-04 T B D T T T D 21.2 3 (8) PM1, BP4 VUS
P8 chr2:241679768 NM_001244008 c.3763G>A p.Val1255Met rs752703226 1.20E-05 D D D T T D D 24.2 6 (8) PM2, PM1, PP2, PP3 LP
P9 chr2:241661285 NM_001244008 c.4682C>T p.Thr1561Met rs769101887 4.00E-06 T B D T T T D 19.77 3 (8) PM2, PM1, BP4 VUS
P10 chr2:241660421 NM_001244008 c.4778C>T p.Pro1593Leu rs200902828 3.00E-04 5.00E-04 D B D T T T D 23.6 4 (8) PM2, PM1, PP2, PP3 LP
P11 chr2:241659285 NM_001244008 c.4927G>A p.Asp1643Asn rs200141437 1.00E-04 3.00E-04 T B D T T T T 18.6 2 (8) PM2, PS4, PM1, PP2, BP4 LP
P12 chr2:241659257 NM_001244008 c.4955G>A p.Arg1652Gln rs376658420 9.70E-05 2.00E-04 T B P T T T’ T 6.526 0 (8) PM2, PM1, BP4 VUS
P13 chr2:241658506 NM_001244008 c.5131G>A p.Asp1711Asn rs199574770 3.20E-05 5.70E-05 D D D T T T D 34 5 (8) PM1, PP3 VUS
P14 chr2:241658487 NM_001244008 c.5150G>T p.Arg1717Leu rs760970824 5.00E-05 2.40E-05 D D D T T D D 35 6 (8) PM2, PM1, PP2, PP3 LP

ALS, amyotrophic lateral sclerosis; SNP, Single-nucleotide polymorphism; ACMG, American College of Medical Genetics and Genomics; VUS, uncertain significance; LP, likely pathogenic; HGMD, Human Gene Mutation Database; SIFT, sorting intolerant from tolerant; PolyPhen2, polymorphism phenotyping version 2; SVM, support vector machine; CADD, combined annotation dependent depletion; M_CAP, Mendelian clinically applicable pathogenicity; SIFT (D: Damaging; T: Tolerable); Polyphen2 (D: Probably_Damaging; P: Possibly_Damaging; B:Benign); Mutation Taster (D: Disease_causing; P: Polymorphism); MetaSVM (T: Tolerable); MetaLR (D: Damaging; T: Tolerable); ClinPred (D: Deleterious; T: Tolerable); M_CAP (D: Damaging; T: Tolerable); CADD: (D: Damaging; T: Tolerable).