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. 2024 Jul 27;16(1):2377576. doi: 10.1080/19490976.2024.2377576

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© 2024 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 8. — AHR-XRE interaction mediated the effect of IAA on Papss2.

(a) The relative mRNA expression levels of Papss2, Slc35b3 in Ahr^−/− mice by quantitative RT-PCR. (b) Protein expression of Papss2 in Ahr^−/− mice by western blot (left panel) and quantification of the value of Papss2/β-actin (right panel). (c) Representative micrographs of immunohistochemical detection of Papss2 (left panel) and quantification of the expression of Papss2 (right panel) in colon tissues of Ahr^−/− mice. Scale bars: 30 µm. (d) CUT&tag assay showed AHR binds to the promoter region of Papss2 and Slc35b3. (e) Logo representation of the motif. ns, not significant, *p < .05, **p < .01.