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. 2024 Jul 29;21:52. doi: 10.1186/s12014-024-09496-3

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Distinct serum proteomic profile in breast cancer patients. (A) Volcano plot showing significantly altered proteins (see text) in serum of progressors at diagnosis (PV1) vs. non-progressors (NP), (B) same as in A, but comparing serum collected at diagnosis (PV1) vs. prior to metastasis within progressors (PV2), (C) Bar plot showing proteins that are enriched for epithelial mesenchymal transition (EMT) comparing PV1 vs. NP, (D) same as in C, but for proteins that are enriched for KEGG pathways describing focal adhesion induced PI3K-Akt signaling, (E) same as in C, but for proteins that are enriched for cell adhesion concept comparing PV1 vs. NP, (F) bar plot showing proteins that were involved in decreased enrichment of cell adhesion proteins comparing serum from PV1 vs. PV2