Figure 8.

A proposed model for OsGRF7 coordinating salinity tolerance and grain size in rice. OsFBO13 interacts with and promotes the degradation of OsGRF7 via the 26S proteasome system. In fbo13 mutant, OsGRF7 bound directly to and activated the expression of OsUGT1/5, and the arbutin content was correspondingly increased. Under salt stress conditions, arbutin enhanced the activities of ROS-scavenging enzymes to eliminate ROS and ultimately induce a salt-tolerance phenotype. At the same time, arbutin also increased the grain size under normal conditions.