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. 2024 Jul 31;46(2):2377785. doi: 10.1080/0886022X.2024.2377785

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© 2024 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 2. — Pathological and ultrastructural changes in the kidneys of the mouse control group and the 16-week diabetic group. (A) HE, masson, PAS staining (×400, scale bar = 20 um) and electron micrographs in renal tissues of db/m group and db/db group. TEM scale bar = 2 um. (B–E) The mesangial area/glomemular area, GBM thickness, foot process width and the number of foot process (/um GBM) were shown using a histogram. Data are presented as mean ± SD; db/m: normal control group; db/db: diabetic group; *p < 0.05, **p < 0.01 db/db vs db/m.