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. 2024 Aug 2;22:94. doi: 10.1186/s12958-024-01254-2

Table 2.

List of candidate genes implicated in mitochondrial dysfunction-associated POI

GENES VARIANTS ACMG pathogenic classification Domain Knockout mice phenotype References
RMND1 c.713 A > G p. (Asn238Ser) P Domain of unknown function DUF155 226–403 NR [160]
c.583G > A p. (Gly195Arg) VUS Nondomain [161]
c.818 A > C p. (Tyr273Ser) LP Domain of unknown function DUF155 226–403 [161]
MRPS22 c.605G > A p. (Arg202His) LP Small ribosomal subunit protein mS22 21–359 Heterozygous MRPS22 knockout mice are fertile and show no overt abnormalities. Homozygous MRPS22 knockout results in embryonic lethality. [124]
c.404G > A p. (Arg135Gln) VUS Small ribosomal subunit protein mS22 21–359 [124]
POLG c.2890 C > T p. (Arg964Cys) VUS Nondomain premature aging and reduced lifespan. [116, 162]
POLG2 c.1297G > T p. (Asp433Tyr) LP Picornavirus capsid 359–522 Heterozygous knockout mice are haplosufficient and develop normally; homozygous knockout mouse is embryonic lethal at day 8.0–8.5 p.c. with concomitant loss of mtDNA and mtDNA gene products. [163, 164]
TFAM c.694 C > T p. (Arg232Cys) VUS HMG-box_TFAM_rpt2 135–234 Embryonic lethal, due to absence of mitochondrial DNA. Mutant embryos die before 10.5 dpc. [119, 165]
LRPPRC c.1061 C > T p. (Ala354Val) P Nondomain Loss of hepatic LRPPRC results in growth delay, and pronounced liver histopathological abnormalities; Loss of LRPPRC induces defective respiration under phosphorylating conditions. [166168]
CLPB c.1159 C > T p. (Arg387Ter) P AAA + ATPase domain 373–527 NR [129]
c.1257 + 5G > A p.? LP AAA + ATPase domain 373–527 [129]
CLPP c.628G > A p. (Ala210Thr) VUS CLP-protease 68–246 Homozygous pups are born at about 60% of the expected Mendelian rate, indicating decreased intrauterine survival. Mutant mice are smaller in size than wild-type littermates, show decreased motor activity, are completely deaf after 12 months and their lifespan is decreased relative to that of wild-type littermates. Both female and male mutant mice are completely infertile due to defects in germ cell development. [132, 169]
TWNK c.1172G > A p. (Arg391His) VUS SF4 helicase domain 384–635 NR [47]
c.1754 A > G p. (Asn585Ser) LP SF4 helicase domain 384–635 [47]
c.1519G > A p. (Val507Ile) VUS SF4 helicase domain 384–635 [47]
c.1321T > G p. (Trp441Gly) LP SF4 helicase domain 384–635 [47]
HARS2 c.1010 A > G p. (Tyr337Cys) VUS Aminoacyl-tRNA synthetase, class II 62–417 Disruption of Hars2in Cochlear Hair Cells Causes Progressive Mitochondrial Dysfunction and Hearing Loss in Mice. [48, 170]
LARS2 c.1565 C > A p. (Thr522Asn) LP Aminoacyl-tRNA synthetase, class Ia 444–600 NR [48]
c.1077delT p.? P Nondomain [48]
c.1886 C > T p. (Thr629Met) VUS Nondomain [48]
TUFM c.524G > C p. (Gly175Ala) VUS Translational (tr)-type GTP-binding domain 58–254 NR [171]
AARS2 c.149T > G p. (Phe50Cys) LP Alanyl-tRNA synthetase, class IIc, core domain 37–792 NR [172]
c.1561 C > T p. (Arg521Ter) P Alanyl-tRNA synthetase, class IIc, core domain 37–792 [172]

Abbreviations: P, pathogenic; LP, likely pathogenic; VUS, variants of uncertain significance; LB: likely benign; B, benign; NR, no record