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. 2024 Aug 2;15:6540. doi: 10.1038/s41467-024-50949-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — LDAH mobilizes stores of esterified sterols that are endogenous LXR ligands and leads to protective modulation of foam cell phenotype, protection against atherosclerosis, and development of more fibrotic and less necrotic lesion architectures. This figure was generated in part using images from Servier Medical Art, under a Creative Commons License CC BY 4.0 (https://creativecommons.org/licenses/by/4.0/).