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. 2024 Jul 22;56(8):1597–1603. doi: 10.1038/s41588-024-01839-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a, Cross-trait genetic correlation of all bleeding in pregnancy phenotypes (with 95% confidence intervals). PPH and early bleeding in pregnancy show no noteworthy genetic correlation. PPH caused by atony or retained placenta are genetically indistinguishable. b, Genetic correlations between PPH and selected disorders. c, Genetic correlations between early bleeding and selected traits. Correlations that are significant after accounting for the number of traits tested are highlighted in yellow text. Error bars, 95% CI. The datasets used for the analysis are described in Supplementary Table 11. HDL, high-density lipoprotein; BMI, body mass index; PTSD, post-traumatic stress disorder.

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