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. 2024 Aug 5;56(8):1592–1596. doi: 10.1038/s41588-024-01844-1

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 9 — In each plot, bars represent the averages across replicates from each experiment (n = 2), points represent the values from the individual replicates (n = 3), and lines show the width of the distribution of the data points. The first column shows mRNA levels of B3GALT5 relative to ACTB transcript in cells treated with four different siRNAs: two targeting B3GALT5 (siRNA1 and siRNA2) and two negative controls (one targeting GAPDH and a scrambled siRNA). The second and third columns show results from infection assay with PR8-GFP (H1N1, multiplicity of infection [MOI] of 0.4 to 10), with the latter column showing infectivity relative to the scrambled siRNA control. The GAPDH siRNA (but not the two siRNAs against B3GALT5) significantly reduced GAPDH expression relative to the scrambled siRNA (~90% reduction). P-values derived from a two-sided Wilcoxon Rank Sum Test and asterisks (*) mark those experiments with P < 0.05.