FIGURE 5.

Mechanisms of neuroprotection exerted by H₂S. H₂S can reduce the infarct size of cerebellar tissue and restore neurological function through mechanisms such as antioxidant, anti‐inflammatory, antiapoptotic, regulating autophagy, protecting mitochondrial function, and vasodilation and generation. By Figdraw. COX‐2, cytochrome oxidase subunit 2; γ‐GCS, γ‐glutamyl cysteine synthetase; iNOS, inducible nitric oxide synthase; mPTP, mitochondrial permeability transition pore; NF‐κB, nuclear factor‐kappa B; VEGF, vascular endothelial growth factor.