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. 2024 Jul 30;103(10):104146. doi: 10.1016/j.psj.2024.104146

Figure 6.

Figure 6

Acrylamide disrupts the network of VIM and prevents its rearrangement to inhibit GAstV-2 replication. (A) Toxicity analysis of different concentrations of acrylamide on GEK cells. (B and C) GAstV-2 mRNA and protein expression levels following acrylamide treatment. (D) The virus titer in the supernatant of acrylamide-treated cells significantly decreased compared with that of the control group. (E) Immunofluorescence colocalization analysis of VIM and GAstV-2 dsRNA. Effect of acrylamide on GAstV-2 induced VIM rearrangement. Immunofluorescence staining for dsRNA, VIM, and DAPI was performed 12 and 24 h after virus infection (scale bars: 15 μm). ns p >0.05, ** p < 0.01, *** p < 0.001.