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. Author manuscript; available in PMC: 2024 Sep 2.
Published in final edited form as: Toxicol Pathol. 2023 Apr 3;51(1-2):39–55. doi: 10.1177/01926233231157322

TABLE 8.

Illumina BaseSpace Correlation Engine generated top canonical pathways common to alveolar/bronchiolar carcinomas (ABCs) from B6C3F1/N mice exposed to Antimony trioxide (CMD) for 2 years and human non-small cell lung cancer as predicted by Broad Institute’s Molecular Signatures Database (MSigDB) using the respective transcriptomic data.

Top 20 concordant canonical pathways in mouse CMD-exposed ABCs and human NSCLC Bioset Score Mouse, −log(p value) Common genes Human, −log(p value) Common genes

Nop56p-associated pre-rRNA complex 93 40.24 76 0.04 18
60S ribosomal subunit, cytoplasmic 68 29.60 42 0.04 8
55S ribosome, mitochondrial 56 24.15 52 17.64 49
40S ribosomal subunit, cytoplasmic 49 21.32 29 0.03 3
Oxidative phosphorylation 48 20.85 60 8.59 52
Huntington’s disease 43 18.49 72 4.47 70
Focal adhesion 40 17.38 65 26.64 97
Vascular smooth muscle contraction 38 16.40 45 20.29 61
Parkinson’s disease 35 15.36 52 5.46 47
39S ribosomal subunit, mitochondrial 35 15.12 32 9.89 29
Validated targets of C-MYC transcriptional activation 35 15.07 41 12.08 41
Cytokine-cytokine receptor interaction 34 14.96 63 19.66 91
Hematopoietic cell lineage 34 14.74 33 12.43 42
Pyrimidine metabolism 29 12.74 42 13.62 49
Alzheimer’s disease 27 11.89 60 3.30 58
Chemokine signaling pathway 25 10.74 49 19.41 83
Purine metabolism 24 10.54 53 11.00 64
LPA receptor mediated events 24 10.48 27 8.92 31
DNA replication 23 10.10 21 11.30 25
28S ribosomal subunit, mitochondrial 23 9.77 20 8.74 20