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Combined in-vivo, ex-vivo, and in-silico approaches were used to separate the contribution of individual remodeling events towards increased MPA pressure. Data from in-vivo and ex-vivo measurements were combined to create an in-silico model of the pulmonary vasculature. CTL: control, PH: pulmonary hypertension, PA: pulmonary artery, MPA: main pulmonary artery, Circ: circumferential, Long: longitudinal, μCT: X-ray microtomography, FSI: fluid structure interaction.
