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. 2024 Aug 5;27(9):1668–1674. doi: 10.1038/s41593-024-01730-3

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 1 — (a) Expression level half violin plots of amyloidogenic pathway genes in all cell types of mouse nervous tissue inputs with SCTransform normalization method from all chosen mouse datasets^9,26,27. (b) Expression level half violin plots of amyloidogenic pathway genes in all cell types of human nervous tissue inputs with SCTransform normalization from all chosen human datasets^28–30. (a, b) Half violins represent aggregated expression levels of respective genes from each cell type and data points refer to individual expression levels from single cells or nuclei normalized by the SCTransform method. (c) Positive rate barplots of amyloidogenic pathway genes in all cell types of mouse nervous tissue inputs from all chosen mouse datasets^9,26,27. (d) Positive rate barplots of amyloidogenic pathway genes in all cell types of human nervous tissue inputs with SCTransform normalization method from all analyzed human datasets^28–30. The results published here are based on data obtained from GEO and the AD Knowledge Portal.

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