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. 2024 May 31;49(11):1738–1748. doi: 10.1038/s41386-024-01884-5

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article–s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article–s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — The first row (A) shows that FC degree was significantly lower after administering alprazolam compared to etifoxine. The second row (B) depicts several regions in which local connectivity, as defined by ReHo, was lower after administering alprazolam compared to etifoxine. The third row (C) shows that low-frequency amplitudes, as derived from fALFF, were higher after administering alprazolam compared to etifoxine. The fourth row (D) illustrates alterations in ICA based resting-state networks, which show higher coherence in the alprazolam condition in the temporal, occipital and right somatosensory cortex. Yellow and blue colours depict t-values (from blue, alprazolam < etifoxine, to yellow, alprazolam > etifoxine).