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. 2024 Jun 9;29(9-10):1291–1308. doi: 10.1007/s10495-024-01988-9

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Enhancement of immunotherapy through ferroptosis in the tumour microenvironment. Ferroptosis is activated or inhibited in tumour cells by different pathways including the following: JAK1-STATA-IRF1 pathway, PGAM1-AKT-LCN2 pathway, APOL3-LDHA-RSL3 pathway, ANO1-PI3K-AKT pathway, and SAPE-OOH-TLR2 pathway, thereby promoting immunotherapy in the TME. This diagram was drawn by Figdraw (www.figdraw.com)