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. 2024 Sep 28;15:8425. doi: 10.1038/s41467-024-52739-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — a, b mRNA and protein of ASPP1 were elevated at 1 week, 2 weeks, and 4 weeks of MI mice. n = 6 independent animals in each group. c mRNA levels of Col1 and ASPP1 in PMCFs treated with TGF-β1 (20 ng/mL), n = 6 independent samples. d Representative immunoblots/densitometric quantitative analysis of Col1 and ASPP1 levels in total lysates of PMCFs treated with TGF-β1 (20 ng/mL). β-actin was used as a loading control, n = 6 independent samples. Data are represented as mean ± SEM. Statistics: one-way ANOVA followed by Tukey post hoc test (a, b). Two-tailed Student’s t-test was used to calculate the presented P values (c, d). Source data are provided as a Source Data file. Ctl control.