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. 2024 Sep 30;15(9):706. doi: 10.1038/s41419-024-07087-6

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Cumulative incidence of bladder cancer for participants with or without autosomal mCAs (A) and mosaic loss (B). Cause-specific hazard models were used to account for the competing risk of death. Model for bladder cancer and model for death from other causes were separately developed, with the same set of variables (as same as model 3 in Table 2) included in both models. Cumulative incidences were estimated and plotted by the status of mCAs and mosaic loss, with all covariates being set to the average level of the study population. mCAs, mosaic chromosomal alterations.