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. 2024 Sep 3;27(10):2021–2032. doi: 10.1038/s41593-024-01742-z

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 1 — a, Age distribution of cohort colored by controls and cases (neurotypical individuals and individuals with a pyschiatric disease) (left), colored by disease classification (middle), and colored by females (F) and males (M) (right) used for the single-nucleus RNA sequencing. b, Boxplots showing (from left to right) age, post-mortem interval (PMI), RNA integrity number (RIN), median number of genes and median number of counts between individuals with a pyschiatric disease (N = 54) and neurotypical individuals (N = 33). The type of test (as indicated in the plots) used was dependent on whether the data was normally distributed (t-test) or not (Mann-Whitney U test); tests were two-sided. Boxplots show the median (center), IQR (bounds of box), and whiskers extending to either the maxima/minima or to the median ± 1.5× IQR, whichever is nearest. Triangles indicate outliers. c, Spearman correlation (two-sided) of age and (from left to right) PMI, RIN, median number of genes and median number of counts.