Fig. 2. FE-SEM images of self-aggregation inhibition of Aβ1–42 after 72 h; (A) a mixture of glycine and NaOH buffer as a blank; (B) formation of plaques of Aβ1–42 only, in the absence of an inhibitor as a control; (C) inhibition of Aβ1–42 plaques with a standard, resveratrol; (D) inhibition of Aβ1–42 plaques with the test compound NS-13.
