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. 2024 Oct 15;21:69. doi: 10.1186/s12979-024-00474-9

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — QC alleviated renal damage in lupus-like mice. (A) Schematics for the experimental workflow with MRL/lpr lupus mice. (B) MRL/lpr lupus mice weight in the QC and control groups. (C) Urine protein changes in the MRL/lpr lupus mice treated for 15 weeks, both in the QC group and the control group. (D) Renal pathological changes from MRL/lpr lupus mice (200-fold field of view; H&E staining). (E) Immune complex deposition in the kidney of lupus-like mice, IgG shown in green, C3 shown in red, nuclei shown in blue (200-fold field of view). (F) Statistical analysis of mean fluorescence intensity of IgG and C3 in glomeruli. (G) Serum total IgG levels, Serum ANA levels and serum dsDNA levels in lupus-like mice. (Quercetin: n = 5, Control: n = 5),(*P < 0.05, **P < 0.01, ****P < 0.0001)