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. 2024 Oct 25;4:215. doi: 10.1038/s43856-024-00648-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 2 — Enzymatic activity for Arylsulfatase-A (ARSA), Arylsulfatase-B (ARSB), N-Sulfoglucosamine Sulfohydrolase (SGSH) and Glycosaminoglycans accumulation (GAG) was assessed in spleen (a–d) liver (e–h), heart (i–l), brain (m–p) and kidney (q–t) tissues, 10 months post-BMT. Activity is represented as percentage of average WT activity. GAG amounts in µg is normalized to µg of DNA content. Recipient and donor Sumf1 genotype is indicated as WT (+/+), or Sumf1^(S153P) homozygous (S153P). Data is represented as box plots with median, min and max. Kruskal-Wallis tests and Dunn’s multiple comparisons test were used. Exact P values are in Supplementary Data 1.