Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2001 Sep;75(18):8690–8696. doi: 10.1128/JVI.75.18.8690-8696.2001

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2001, American Society for Microbiology

PMC Copyright notice

FIG. 3 — Control of fatal SIV-related tuberculosis-like disease in SIV/BCG-coinfected macaques correlated temporally with the restoration of BCG PPD-specific T-cell responses during the period of antiretroviral treatment. The data shown in panel a were generated in proliferation assays, using PBL from the coinfected macaques depleted of CD8⁺ lymphocytes. (b) Correlation between the resolution of BCG infection and the restored T-cell responses to BCG after antiretroviral treatment. Follow-up studies showed that the ability of T cells to proliferate was suppressed again due to the rebound SIV infection after the discontinuation of antiretroviral treatment (data not shown).

FIG. 3 — Control of fatal SIV-related tuberculosis-like disease in SIV/BCG-coinfected macaques correlated temporally with the restoration of BCG PPD-specific T-cell responses during the period of antiretroviral treatment. The data shown in panel a were generated in proliferation assays, using PBL from the coinfected macaques depleted of CD8⁺ lymphocytes. (b) Correlation between the resolution of BCG infection and the restored T-cell responses to BCG after antiretroviral treatment. Follow-up studies showed that the ability of T cells to proliferate was suppressed again due to the rebound SIV infection after the discontinuation of antiretroviral treatment (data not shown).