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. 2024 Jun 6;109(11):3615–3630. doi: 10.3324/haematol.2023.284332

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Copyright© 2024 Ferrata Storti Foundation

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Figure 2. — The mutational landscape of non-Hodgkin lymphoma differs by Epstein-Barr virus and immune status. (A) Co-oncoplot of the most recurrently mutated genes (≥15%) within the 49 samples of large B-cell lymphoma (LBCL) according to Epstein-Barr virus (EBV) status (EBV^– on the left, EBV⁺ on the right). TP53, MYD88 and HNRNFP were differently mutated between the two groups but these results lost their significant value after false discovery rate (FDR) correction was applied. (B) Co-oncoplot of the most recurrently mutated genes (≥15%) within the 49 LBCL samples according to immune status (immunocompetent on the left, immunodeficient on the right). STAT 3, TYW1 and MYD88 were differently mutated between the two groups but these results lost their significant value after FDR correction was applied. Fisher exact test was used to compare categorical data. CNS: central nervous system; PTLD: post-transplant lymphoproliferative disorder; DLBCL: diffuse large B-cell lymphoma; ID: immunodeficient; IC: immunocompetent; HIV: human immunodeficiency virus.