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. 2024 Nov 14;14:28055. doi: 10.1038/s41598-024-78462-8

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — CD62P on platelets is upregulated in patients with UC. Expression of platelet activation markers in patients with UC was compared with that in healthy controls using flow cytometry. (a) Dot plots represent the platelet gating strategy. Platelets were identified in whole blood according to their FSC/SSC and CD61 positivity characteristics. (b) Representative patterns of expression of CD62P, CD40L, CD63, PAC1, annexin V, and CD36 on platelets in patients with UC and healthy controls. (c) Expression of the markers shown in (b) in patients with UC (n = 54) was quantified and compared with that in healthy controls (n = 11). The bar graphs represent the median expression with the IQR. P-values were calculated using the Mann–Whitney U test; *P < 0.05. UC, ulcerative colitis; FSC, forward scatter; SSC, side scatter; IQR, interquartile range.