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. 2024 Aug 16;2(11):776–785. doi: 10.1021/envhealth.4c00098

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© 2024 The Authors. Co-published by Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences, and American Chemical Society

Permits non-commercial access and re-use, provided that author attribution and integrity are maintained; but does not permit creation of adaptations or other derivative works (https://creativecommons.org/licenses/by-nc-nd/4.0/).

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Effects of F-53B exposure on the PKA/CREB pathways in fetal mice and HT22 neurons. (A-B) The relative mRNA levels of the PKA/CREB pathways related genes in the fetal brain and HT22 neurons. (C-E) Representative photomicrographs of PKA levels and location in HT22 neurons. (F-I) The blots and diagrams of the PKA/CREB pathways related protein in the nucleus and cytoplasm of HT22 neurons. Comparison with the control group is indicated by the symbol “*”. Comparison with the 10 μmol/L F-53B-exposed group is indicated by the symbol “^&”. * and ^& indicate p < 0.05, ** and ^&& indicate p < 0.01, *** and ^&&& indicate p < 0.001.