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. 2024 Nov 19;14:28578. doi: 10.1038/s41598-024-79367-2

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 2 — Overexpression of AP001007 improved the viability and mitochondrial function in LPS-treated HK-2 cells. (A) qPCR data showing the expression of AP001007 in HK-2 cells with the indicated treatments. (B) MTT assay results depicting the viability of HK-2 cells with the indicated treatments. (C) JC-1 staining outcome illustrating the mitochondrial membrane potential in HK-2 cells with the indicated treatments. (D) The quantification results of data shown in (C). Mock defines PBS treatment and vector defines transfection with empty backbone plasmid for AP001007 overexpression (OE).