Skip to main content
. 2024 Nov 21;16:165. doi: 10.1186/s13148-024-01785-w

Table 2.

Potential clinical significance of histone posttranslational modifications in HNSCC

PTMs Inhibitor Function References
Acetylation HATs CCS1477 Phase I/II clinical studies for the treatment of hematologic malignancies, prostate cancer, breast cancer and non-small cell lung cancer have been initiated [83]
EP31670 Phase I clinical trial of safety and maximum tolerated dose in advanced solid tumors
HDACs SAHA

1. Treatment of cutaneous T-cell lymphoma, laryngeal squamous cell carcinoma

2. Inhibits cell proliferation, induces cell cycle arrest and apoptosis

3. The combination with pembrolizumab has stronger therapeutic activity against HNSCC

[86]

[88]

[92]

Romidepsin

1. Treatment of cutaneous T-cell lymphoma

2. Inhibits tumor-associated histone deacetylase

[83]
Valproic acid

1. Increase cell arrest and promote apoptosis

2. It has strong efficacy in combination with cisplatin

[93]
TSA

1. By regulating cell cycle mediators

2. Inhibits cell proliferation and induces G2/M phase arrest in tumor cells

3. TSA-induced H3K9ac can ameliorate the chemoresistance of HNSCC to cisplatin under active NF-κB signaling pathway

[94]

[95]

PBA

1. Damage to the EMT process

2. It increased p21 and p27, decreased the levels of G1/S molecules CDK6, cyclin D1 and phosphorylated kinase, and inhibited the growth of oral squamous cell carcinoma

[96]
[97]
Methylation HMTs Chaetocin It plays an important role in the combination of drugs in cancer treatment [83]

BIX-01294

CUDC-101

Inhibits H3K9me2 expression

Phage I study on intermediate or high-risk HNSCC patients

[103]

[113]

GSK-343

1. The tumor growth of tongue squamous cell carcinoma was significantly inhibited after DZNeP administration

2. DZNeP is effective against a variety of cancers, including HNSCC, either alone or in combination

3. Associated with HPV status

[104]

[105]

[106]

DZNeP
EPZ-568
HNMTs TCP Most are associated with hematologic disorders [83]
ORY-1001
INCB059872
IMG-7289
TAK-418
GSK-LSD1

1. Blocks EGF-induced proliferation

2. Weakens genes involved in carcinogenic properties

Inhibits the growth of oral squamous cell carcinoma

[112]