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. 2024 Sep 15;38(12):2614–2627. doi: 10.1038/s41375-024-02394-5

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A CEP53 cells in suspension (far left), Wright-Giemsa staining of a cytospin slide of CEP53 cells (left) and flow cytometric analysis of CEP53 cells for GFP (right) and Ter119 and CD71 (far right). B CEP53 cells were transplanted into non-irradiated recipient mice. Bone marrow (BM) and spleen (SPL) cells were collected from two moribund mice. Expression of GFP, Ter119, CD71 in BM and SPL cells were shown. Note that almost all GFP⁺ CEP53 cells were CD71⁺Ter119^+/− erythroblasts.