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. 2024 May 31;16(2):137–145. doi: 10.4274/jcrpe.galenos.2023.2023-5-10

Table 3. Clinical characteristics of 13 patients diagnosed with autoantibody-negative type 1 diabetes mellitus and genetic identification with variants of uncertain significance detected according to According to the American College of Medical Genetics and Genomics.

 No

MODY gene

Age at diagnosis (year)

Gender

Family history for MODY/generations

Clinical presentation

HbA1c % level at diagnosis

Follow-up treatment

1

HNF1A

10.1

Male

+/3

Hyperglycaemia

8.4

Diet, sulfonylurea

2

HNF1A

14

Female

+/3

Hyperglycaemia

7.1

Diet, sulfonylurea, insulin

3

HNF1B

4.0

Male

-/1

Diabetic ketoacidosis

8.9

Insulin

4

HNF1B

8.3

Female

NA

Diabetic ketoacidozis

9.4

Insulin

5

HNF4A

2.3

Male

+/3

Hyperglycaemia

6.9

Diet, sulfonylurea, insulin

6

BLK

1.5

Male

+/2

Hyperglycaemia

7.4

Diet, sulfonylurea, insulin

7

BLK

10

Female

No

Hyperglycaemia

10.2

Diet, sulfonylurea, insulin

8

PAX4

5.5

Female

+/2

Diabetic ketoacidosis

8.8

Diet, sulfonylurea, insulin

9

INS

10.5

Female

+/2

Diabetic ketoacidosis

9.2

Diet, diet, sulfonylurea, insulin

10

KLF11

8.5

Male

+/2

Diabetic ketosis

7.7

Diet, sulfonylurea, insulin

11

ABCC8

2.4

Male

+/2

Hyperglycaemia

7.1

Sulfonylurea

12

ABCC8

4.2

Male

+/2

Hyperglycaemia

7.3

Sulfonylurea

13

RFX6

2.4

Male

No

Diabetic ketosis

7.4

Diet, sulfonylurea, insulin

OADs: oral antidiabetic drugs, NA: not available, HbA1c: glycated hemoglobin