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. 2024 May 31;6(6):1108–1127. doi: 10.1038/s42255-024-01053-4

Fig. 7. Pyridoxal and supersulfides reverse enhanced inflammatory response of macrophages in prolonged hypoxia.

Fig. 7

a, Lysosomal acidification in BMDMs. Representative AcidiFluor ORANGE staining (left) and its quantification (right) from three independent experiments. CHyp + Pyridoxal, BMDMs differentiated under 1% oxygen in the presence of 50 μg ml−1 pyridoxal. b. Immunoblot analysis detecting Lamp1 in BMDMs. Tubulin was detected as a loading control. A representative result from three independent experiments is shown. c, Expression of representative lysosome-related genes in BMDMs (n = 3 biologically independent samples). d, Immunoblot analysis detecting 2OG-dependent dioxygenases in BMDMs. Tubulin was detected as a loading control. A representative result from three independent experiments is shown. e, Relative Il6 expression in BMDMs (n = 3 biologically independent samples). f, ELISA of IL-6 in the culture supernatant of BMDMs (n = 4 biologically independent samples). g, Il6 expression in BMDM with or without PNPO overexpression (n = 4 biologically independent samples). h, SSP4 staining to detect supersulfides in BMDM. Representative SSP4 staining (left) and quantification (right) from three independent experiments. i, Effects of GSSSG, a supersulfide donor, and GSSG on lysosomal acidification. Representative AcidiFluor ORANGE staining (left) and its quantification (right) from three independent experiments. Oxidized glutathione, GSSG, was added as a negative control. j, Il6 expression in BMDM treated with GSSSG or GSSG (n = 3 biologically independent samples). Scale bars, 50 μm (a,h,i). Error bars represent s.e.m. (c,eg,j). One-way ANOVA (a,h) and two-way ANOVA (c,eg,i,j) were conducted to evaluate statistical significance.

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