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. 2024 Nov 12;15:1480676. doi: 10.3389/fimmu.2024.1480676

Table 3.

Differences among groups in baseline characteristics.

NEDA-3
(n = 85)
INFL
(n = 20)
PIRA
(n = 10)
p-Value NEDA-3 versus INFL p-Value NEDA-3 versus PIRA p-Value INFL versus PIRA
Age (years)
Median [range]
42.4
[22.1–76.9]
37.3
[25.2–61.4]
44.9
[28.9–62.7]
ns ns ns
BMI
Median [range]
23.1
[16.7–37.9]
23.8
[18.5–28.8]
21.3
[15.4–29.9]
ns ns ns
Sex (F/M) 51/34 18/2 7/3 0.02 ns ns
Current smoker
N (%)
9 (10.6%) 3 (15.0%) 0 (0.0%) ns ns ns
Disease duration
Median [range]
7.4
[0.2–28.3]
4.9
[0.1–18.0]
10.7
[1.6–30.8]
ns ns ns
EDSS score
Median [range]
2.5
[0.0–6.5]
2.0
[1.5–6.0]
3.3
[1.0–6.0]
ns ns ns
sNfL
Median [range]
7.2
[1.6–120.7]
16.0
[2.2–382.9]
6.7
[2.5–16.7]
0.0006 ns 0.01
z-score sNfL
Median [range]
−0.1
[−3.4–3.5]
2.5
[−3.0–4.0]
−0.3
[−3.2–1.0]
0.0003 ns 0.01
sGFAP
Median [range]
126.4
[48.6–1,285.1]
199.1
[85.3–1,105.8]
154.5
[75.1–417.2]
0.03 ns ns
Card. Comorbidities N (%)
High blood pressure
Cardiomyopathy
Ischemic heart disease
5 (5.9%)
0 (0.0%)
0 (0.0%)
2 (10.0%)
1 (5.0%)
0 (0.0%)
0 (0.0%)
0 (0.0%)
0 (0.0%)
ns ns ns
Previous treatment N (%)
Naïve
Platform
Orals
Monoclonal Ab.
19 (22.4%)
23 (27.1%)
26 (30.5%)
17 (20.0%)
8 (40.0%)
4 (20.0%)
7 (35.0%)
1 (5.0%)
0 (0%)
0 (0%)
10 (100%)
0 (0%)
ns 0.0004 0.009
Reason for switching previous treatment N (%)
Effectiveness
Safety
58 (87.9%)
8 (12.1%)
11 (91.6%)
1 (8.4%)
9 (90%)
1 (10%)
ns ns ns
T2 lesions on MRI N (%).
<10 lesions
10–50 lesions
50–100 lesions
>100 lesions
9 (10.6%)
59 (69.4%)
16 (18.8%)
1 (1.2%)
3 (15.0%)
11 (55.0%)
4 (20.0%)
2 (10.0%)
0 (0.0%)
7 (70.0%)
3 (30.0%)
0 (0.0%)
ns ns ns
Gd-lesions on MR N (%).
At least one lesion 35 (41.2%) 12 (60.0%) 2 (20.0%) ns ns ns

BMI, body mass index; F, female; M, male; Card, cardiovascular; EDSS, Expanded Disability Status Scale; sNfL, serum neurofilament light chains; sGFAP, serum glial fibrillary acidic protein; n, number of patients; NEDA-3, patients free of disease activity at 12 months of ocrelizumab initiation (n = 85); INFL, patients with new relapses and/or radiological activity during follow-up (n =2 0); PIRA, patients who had confirmed disability progression in the absence of relapses or new MRI activity at 12 months of ocrelizumab initiation (n = 10); ns, non-significant; N, number of patients; Ab, antibody; Naïve, no previous treatment; Gd, gadolinium-enhancing lesions.

Platform treatments included interferon beta and glatiramer acetate. Oral drugs included cladribine, dimethylfumarate, fingolimod, and teriflunomide. Monoclonal Ab. included alemtuzumab and natalizumab.