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. 2024 Nov 27;24:409. doi: 10.1186/s12906-024-04711-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A histogram showing serum reproductive hormones levels and means of fold changes of hormone receptor genes in testicular tissue collected from all rats in the four groups. A Testosterone T, (B) Follicle-stimulating hormone FSH, (C) Luteinizing hormone LH, and (D) Follicle-stimulating hormone receptor (FSHR), luteinizing hormone receptor (LHR) and androgen receptor (AR). G1: Control, G2: Anacyclus extract treated group, G3: Cadmium treated group, G4: Cadmium and extract treated group. Data is presented as mean ± standard error (S.E.). Different letters represent significant differences (P ≤ 0.05) between experimental groups