TABLE 5.
Count | First author | Journal | Title | Year | Citations |
---|---|---|---|---|---|
24 | Alicic RZ | Clin J Am Soc Nephro | Diabetic Kidney Disease Challenges, Progress, and Possibilities | 2017 | 1,142 |
22 | Li YJ | Am J Pathol | Epithelial-to-mesenchymal transition is a potential pathway leading to podocyte dysfunction and proteinuria | 2008 | 312 |
20 | Chen L | Chem-Biol Interact | Role of RAS/Wnt/beta-catenin axis activation in the pathogenesis of podocyte injury and tubulo-interstitial nephropathy | 2017 | 80 |
20 | Meng XM | Nat rev Nephrol | TGF-beta: the master regulator of fibrosis | 2016 | 1868 |
20 | Zhou LL | Nat Rev Nephrol | Wnt/beta-catenin signalling and podocyte dysfunction in proteinuric kidney disease | 2015 | 164 |
19 | Hu MS | J Cell Mol Med | LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose-induced podocyte injury via its interplay with beta-catenin | 2017 | 160 |
19 | Wang M | Phytomedicine | Poricoic acid ZA, a novel RAS inhibitor, attenuates tubulo-interstitial fibrosis and podocyte injury by inhibiting TGF-beta/Smad signaling pathway | 2017 | 82 |
19 | Wiggins RC | Kidney Int | The spectrum of podocytopathies: A unifying view of glomerular diseases | 2007 | 606 |
18 | Liu YH | J Am Soc Nephrol | New Insights into Epithelial-Mesenchymal Transition in Kidney Fibrosis | 2010 | 768 |
18 | Shankland SJ | Kidney Int | The podocyte’s response to injury: Role in proteinuria and glomerulosclerosis | 2006 | 718 |