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. 2024 Nov 29;15:289. doi: 10.1186/s13244-024-01859-6

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Overview of study design and analyses. We recruited a total of 276 participants (CD, n = 230; HCs, n = 46) and collected fecal samples, blood samples, multiparameter brain MRI data, and responses to a psychological questionnaire. Subsequently, a multiomics analysis of the Gut-brain axis was conducted to investigate the brain features of CD and the underlying mechanisms. (CD, Crohn’s disease; HCs, healthy controls; 16Sr RNA, 16S ribosomal ribonucleic acid)