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. 2024 Nov 29;14:29720. doi: 10.1038/s41598-024-80986-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 4 — Two-sample MR analysis results reveals the association between Glu levels and PCa risk. A Forest plot displaying MR estimates and 95% confidence intervals for the causal effect of Glu levels on PCa risk. B Scatter plot depicting the relationship between genetically predicted Glu levels and the risk of PCa using different MR methodologies (inverse-variance weighted, MR-Egger, and weighted median). C Leave-one-out sensitivity analysis indicating the stability of the association between Glu and PCa risk, demonstrating that no single SNP disproportionately influenced the results.