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. 2024 Nov 30;14:29813. doi: 10.1038/s41598-024-81352-8

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — Plasma exosomal DEmiRNAs identified by small RNA-Seq in patients with SCAD and the corresponding GO (cellular component) enrichment analysis. (A) Plasma exosomal statistically significant DEmiRNAs in patients with SCAD (n = 3) in comparison with healthy controls (n = 3). DEmiRNAs up-regulated and down-regulated with statistical significance are represented by the red and green points, respectively. (B) Enrichment analysis of GO (cellular component) presenting the most abundant cellular components associated with these DEmiRNAs target genes. no-DEGS: not differentially expressed genes; SCAD: stable coronary artery disease; GO: Gene Ontology; DEmiRNAs: differentially expressed miRNAs.