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. 2024 Dec 4;7:1617. doi: 10.1038/s42003-024-07289-w

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — a Schematics of the experimental design for single-cell RNA (sc-RNA) sequencing. Peripheral blood mononuclear cells (PBMCs) were processed via sc-RNA sequencing using the 10x-Based Genomics platform after collected from herpes zoster patients (HP), herpes zoster patients after recovery (RP) and healthy controls (HA). b UMAP plot showing eight subpopulations of PBMCs including neutrophils, MPs, plasma cells, T cells, B cells, basophils, CDCs, platelets and erythrocytes identified using integrated and classification analysis. c The heatmaps show differentially expressed genes (DEGs) upregulated in eight subpopulations of PBMCs. d UMAP projection of canonical markers, including CD3E, CD4, CD8A, and NCAM1 for NK and T cells; CD14, CD1C, and FCGR3A for myeloid cells; and CD19 for B cells as indicated in the legend.