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. 2024 Nov 26;11:1486503. doi: 10.3389/fmed.2024.1486503

Table 2.

Distributions of patients’ characteristics among complete cases and multiple imputation.

Complete case (N = 288) Multiple imputation (N = 369)
Female 150 (52.1%) 188 (50.9%)
Age (years) 49.0 (42.0–56.0) 49.0 (42.0–56.0)
Age ≥ 50 years 138 (47.9%) 181 (49.1%)
Liver cirrhosis 23 (8.0%) 36 (9.8%)
HCV RNA(lg10 IU/L) 6.0 (5.2–6.4) 5.9 (5.2–6.5)
HCV Genotype
1 120 (41.7%) 154 (41.7%)
2 21 (7.3%) 28 (7.6%)
3 112 (38.9%) 138 (37.4%)
6 26 (9.0%) 37 (10.0%)
Unknown 9 (3.1%) 12 (3.3%)
Total bilirubin (μmol/L) 12.5 (9.5–16.8) 12.6 (9.4–17.2)
Alanine aminotransferase (×ULN) 1.4 (0.9–2.4) 1.4 (0.9–2.5)
Alanine aminotransferase >2 × ULN 100 (34.7%) 130 (35.2%)
Albumin (g/L) 44.3 (41.1–47.0) 44.2 (40.8–46.8)
Serum creatinine (×ULN) 0.6 (0.5–0.7) 0.6 (0.5–0.7)
Platelet (×109/L) 144.0 (103.0–189.5) 144.0 (100.5–189.0)
Platelet >100 × 109/L 223 (77.4%) 277 (75.1%)
SII 238.2 (153.0–358.2) 228.3 (146.6–345.8)
Ribavirin administration 108 (37.5%) 135 (36.6%)
SVR12 287 (99.7%) 366 (99.2%)
Outcome of liver fibrosis
Non-improved APRI 239 (83.0%) 299 (81.0%)
Non-improved FIB-4 252 (87.5%) 323 (87.5%)

SII, systemic immune-inflammation index; HCV, hepatitis C virus; ULN, upper limit of normal; SVR12, HCV RNA less than 15 IU/mL 12 weeks after cessation of antiviral therapy; FIB-4, fibrosis-4; APRI, aspartate aminotransferase-to-platelet ratio index. Quantitative data are presented as median (interquartile range). Qualitative data are presented as frequencies (proportions).