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. 2024 Dec 10;14:29371. doi: 10.1038/s41598-024-80511-1

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — Effect of met-hemoCD-I antidotes on mice poisoned by NaSH. (A) Lactate levels in the blood of mice treated with NaSH followed by met-hemoCD-I. (B–D) Cytochrome c oxidase (CcO) activity in the brain (B), heart (C), and liver (D) of mice treated with NaSH followed by met-hemoCD-I. In the model, a solution of NaSH (21 mg/kg) in PBS was intraperitoneally injected into mice, followed by an intraperitoneal injection of met-hemoCD-I (7 mM, 0.2 mL). Each bar represents the mean ± SE (n ≧ 3). The asterisks denote statistical significance, ***P < 0.005. **P < 0.01, *P < 0.05.