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. 2024 Jul 10;631(8021):654–662. doi: 10.1038/s41586-024-07623-6

Extended Data Fig. 11. Erastin quantitatively modulates ROS feedback loops.

Extended Data Fig. 11

a, ROS steady state remains low regardless of photoinduction intensity in the absence of erastin treatment. Single-cell ROS steady states were measured in cells (40 cells) treated with or without erastin (10 µM) after photoinduction with different light intensities (1: 60 mW for 10 s; 2: 240 mW for 10 s; 3: 240 mW for 40 s). b-e, Cellular levels of GSH (b), labile iron (c), NOX activity (d), and ROS (e) after treatment with different erastin concentrations. Data represent mean ± s.d. (GSH: four technical replicates; labile iron: ≥ 257 cells; NOX activity: three biological repeats, with three technical repeats each; and ROS: ≥ 249 cells). Measurements are significantly different from those of untreated (0 µM) cells (two-sided Wilcoxon rank-sum tests, * FDR-adjusted P < 0.05 (b), <2 × 10−30 (c), <4 × 10−3 (d), <2 × 10−5 (e)). (f) Representative images of ROS (upper panels) and labile iron (lower panels) 8 h after different erastin treatments. Scale bar, 50 μm. All experiments were independently repeated three times with similar trends.

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