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. 2024 Dec 18;15:762. doi: 10.1007/s12672-024-01562-4

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 7 — Transcriptional alterations and expression of MDD marker genes in TNBC disease and enrichment pathway analysis of risk genes. A Localization of CNV alterations on MDD marker genes on chromosome 23. B Correlation study of the expression of 10 MDD marker genes (C) Correlation visualization plot of 10 MDD marker genes in TNBC disease. D Principal component analysis showing significant differences between MDD marker high-risk and low-risk cohorts (E) Heatmap showing differences in clinical information and expression between MDD marker high-risk and low-risk cohorts (F) Regulatory network map of miRNA-transcription factors for 10 key genes. G Bubble diagram showing the results of KEGG enrichment analysis. H GSEA analyses of different KEGG pathways were clustered in the high-risk and low-risk groups. *p ≤ 0.5; **p ≤ 0.01; ***p ≤ 0.001; ****p ≤ 0.0001