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. 2024 Dec 18;21:107. doi: 10.1186/s12986-024-00883-z

Table 1.

Characteristics of skeletal muscle nitric oxide synthase (NOS) isoforms [37, 42, 44, 50, 54, 64, 65]

nNOS variants eNOS iNOS
Localization Sarcolemma (nNOSμ) Plasma caveoli of ECs Sarcolemma
Sarcoplasm (nNOSμ) Mt
Golgi apparatus (nNOSβ)
Mt (nNOSα)
Co-localized molecules α1-syn (sarcolemmal nNOSμ) Cav-1 Cav-3
SR, RyR1 (sarcoplasmic nNOSμ) Mt-complex II
sGC, PKG (nNOSβ)
PGC1α, Mt-complex I, IV (nNOSα)`
Up-regulators Hypoxia, crush injury, contraction, myotube fusion Hypoxia, contraction, HSP Cytokines, myotube fusion
Inhibitors Cav-3, PIN protein Cav-1 Kalirin, Cav-3
Post-translational modification Phosphorylation, nitrosylation Phosphorylation, nitrosylation, myristoylation, palmitoylation Phosphorylation, nitrosylation

α1-syn, α1-syntrophin; Cav; caveolin; ECs, endothelial cells; eNOS, endothelial nitric oxide synthase; HSP, heat shock protein; iNOS; inducible nitric oxide synthase; Mt, mitochondria; nNOS, neuronal nitric oxide synthase; PIN, protein inhibitor of nitric oxide synthase; PGC-1α, peroxisome proliferator activated receptor-γ co-activator 1α; PKG, protein kinase G; RyR1; ryanodine receptor 1 Ca2+ release channel; sGC; soluble guanylate cyclase; SR, sarcoplasmic reticulum